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- 01GKT5NKN73569M104FT5RT7S5 classification A1.
- 01GKT5NKN73569M104FT5RT7S5 date "2022".
- 01GKT5NKN73569M104FT5RT7S5 language "eng".
- 01GKT5NKN73569M104FT5RT7S5 type journalArticle.
- 01GKT5NKN73569M104FT5RT7S5 hasPart 01GKT5ZM4448GVVY7ZND2XE9DN.pdf.
- 01GKT5NKN73569M104FT5RT7S5 subject "Biology and Life Sciences".
- 01GKT5NKN73569M104FT5RT7S5 subject "Medicine and Health Sciences".
- 01GKT5NKN73569M104FT5RT7S5 doi "10.1155/2022/8078259".
- 01GKT5NKN73569M104FT5RT7S5 issn "2314-6133".
- 01GKT5NKN73569M104FT5RT7S5 issn "2314-6141".
- 01GKT5NKN73569M104FT5RT7S5 volume "2022".
- 01GKT5NKN73569M104FT5RT7S5 abstract "Coronaviruses are a family of viruses that infect mammals and birds. Coronaviruses cause infections of the respiratory system in humans, which can be minor or fatal. A comparative transcriptomic analysis has been performed to establish essential profiles of the gene expression of Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) linked to cystic fibrosis (CF). Transcriptomic studies have been carried out in relation to SARS-CoV-2 since a number of people have been diagnosed with CF. The recognition of differentially expressed genes demonstrated 8 concordant genes shared between the SARS-CoV-2 and CF. Extensive gene ontology analysis and the discovery of pathway enrichment demonstrated SARS-CoV-2 response to CF. The gene ontological terms and pathway enrichment mechanisms derived from this research may affect the production of successful drugs, especially for the people with the following disorder. Identification of TF-miRNA association network reveals the interconnection between TF genes and miRNAs, which may be effective to reveal the other influenced disease that occurs for SARS-CoV-2 to CF. The enrichment of pathways reveals SARS-CoV-2-associated CF mostly engaged with the type of innate immune system, Toll-like receptor signaling pathway, pantothenate and CoA biosynthesis, allograft rejection, graft-versus-host disease, intestinal immune network for IgA production, mineral absorption, autoimmune thyroid disease, legionellosis, viral myocarditis, inflammatory bowel disease (IBD), etc. The drug compound identification demonstrates that the drug targets of IMIQUIMOD and raloxifene are the most significant with the significant hub DEGs.".
- 01GKT5NKN73569M104FT5RT7S5 author f9fd1efc-4754-11ed-876b-8621c6d3f5e8.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:3bbf0c83-2c86-4e2c-b462-88875f85773f.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:7f5ca89c-6fe3-4855-b10f-8bc2c931fddc.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:8a5b6039-2847-4476-9a46-1170a5923c6c.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:a5d5be8d-e269-4568-af9b-82755c888a7b.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:ad60a93b-59f9-4f74-bcae-ca1019361474.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:cab27428-1dd9-4173-8016-0b07c41f201f.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:ee0fce8a-8798-4cfd-b221-21173862a523.
- 01GKT5NKN73569M104FT5RT7S5 author urn:uuid:ef5958ea-6198-4ad3-a86b-2ab88a95d719.
- 01GKT5NKN73569M104FT5RT7S5 dateCreated "2022-12-09T00:48:22Z".
- 01GKT5NKN73569M104FT5RT7S5 dateModified "2024-10-29T17:42:30Z".
- 01GKT5NKN73569M104FT5RT7S5 editor urn:uuid:b9279f02-3a5b-49cc-80e7-8fe8ed068d00.
- 01GKT5NKN73569M104FT5RT7S5 name "Discovering common pathophysiological processes between COVID-19 and cystic fibrosis by differential gene expression pattern analysis".
- 01GKT5NKN73569M104FT5RT7S5 pagination urn:uuid:35dbb1d7-7ed1-40db-ae8d-b4560bd540a0.
- 01GKT5NKN73569M104FT5RT7S5 publisher urn:uuid:bbf288d4-f096-448f-b69a-f941b5479ac8.
- 01GKT5NKN73569M104FT5RT7S5 sameAs LU-01GKT5NKN73569M104FT5RT7S5.
- 01GKT5NKN73569M104FT5RT7S5 sourceOrganization urn:uuid:55af027e-dc76-4c16-b7e0-9c8b72212f91.
- 01GKT5NKN73569M104FT5RT7S5 type A1.