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- aggregation classification "C3".
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation date "2010".
- aggregation hasFormat 1068617.bibtex.
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- aggregation isPartOf urn:issn:1129-2369.
- aggregation language "eng".
- aggregation subject "Medicine and Health Sciences".
- aggregation title "More antisaccade errors in migraine patients".
- aggregation abstract "Aim Recent voxel-based morphometry studies in migraine patients have shown subtle but significant reductions in grey matter in cortical areas involved in pain processing (including the prefrontal and cingulate cortices), which are related to migraine frequency. (1-5) We hypothesized these changes would be reflected in the control of saccadic eye movements. Methods Saccades were recorded by infrared-oculography. Three tasks were performed: a prosaccade gap and overlap task during which the subject was instructed to look at the stimulus, and an antisaccade gap task during which the subject had to look in the opposite direction of the stimulus. Latency and direction were analyzed. These parameters were compared between patients with migraine (n = 80) and controls (n = 87). Results Our results suggested a greater latency variability in migraine patients (pro-gap P = 0.002 and pro-overlap P = 0.004). The latency of the prosaccades with gap was borderline increased in migraine (P = 0.042). Migraine patients on prophylactic therapy made significantly more directional errors than controls in the antisaccade gap task (P = 0.001). The group on prophylaxis had on average 10.66 days migraine per month, those who did not take prophylactic medication had on average 3.85 migraine days a month. No significant differences were found between migraine patients with and without aura for all parameters studied. Conclusion We found abnormal saccade behaviour in migraine patients, especially more antisaccade errors in patients on prophylactic drugs, suggesting it is related to migraine frequency. We hypothesize that grey matter changes in the prefrontal and cingulate cortex account for this abnormal antisaccade behaviour. 1. Cephalalgia 2008;28(6):598-604. 2. Cephalalgia 2008;28(1):1-4. 3. Headache 2008;48:109-17. 4. Cephalalgia 2009;30(1):53-61. 5. Ann N Y Acad Sci 2005;1039:239-51.".
- aggregation authorList BK215418.
- aggregation endPage "S52".
- aggregation issue "suppl. 1".
- aggregation startPage "S52".
- aggregation volume "11".
- aggregation isDescribedBy 1068617.
- aggregation similarTo LU-1068617.