Matches in UGent Biblio for { <https://biblio.ugent.be/publication/295171#aggregation> ?p ?o. }
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- aggregation classification "A1".
- aggregation creator B143862.
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation date "2004".
- aggregation format "application/pdf".
- aggregation hasFormat 295171.bibtex.
- aggregation hasFormat 295171.csv.
- aggregation hasFormat 295171.dc.
- aggregation hasFormat 295171.didl.
- aggregation hasFormat 295171.doc.
- aggregation hasFormat 295171.json.
- aggregation hasFormat 295171.mets.
- aggregation hasFormat 295171.mods.
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- aggregation hasFormat 295171.xls.
- aggregation hasFormat 295171.yaml.
- aggregation isPartOf urn:issn:0730-7268.
- aggregation language "eng".
- aggregation rights "I have transferred the copyright for this publication to the publisher".
- aggregation subject "Biology and Life Sciences".
- aggregation title "Testosterone and energy metabolism in the estuarine mysid Neomysis integer (Crustacea: Mysidacea) following exposure to endocrine disruptors".
- aggregation abstract "A diverse set of reference compounds suspected of having, an endocrine-disrupting mode of action (i.e., testosterone, flutamide, ethinylestradiol, precocene, nonylphenol, fenoxycarb, and methoprene) were tested for acute toxicity to the estuarine mysid Neomysis integer (Crustacea: Mysidacea). Neomysis integer was very sensitive to all tested compounds, with 96-h median lethal concentrations in a narrow range between 0.32 and 1.95 mg/L. The pesticides methoprene and fenoxycarb, both synthetic insect juvenile hormone analogs, were most toxic to N. integer. In addition, the short-term sublethal effects of methoprene and nonylphenol (an estrogen agonist) on the energy and steroid metabolism of N. integer were evaluated. Both compounds significantly affected energy and testosterone metabolism of N. integer at concentrations, below acute toxicity levels. Energy consumption in methoprene- and nonylphenol-exposed mysids was significantly induced at 100 mug/L, resulting in a lower cellular energy allocation in these animals. Testosterone phase I metabolism was affected at 10 mug/L, whereas glycosylation was the most important phase 11 pathway affected in mysids exposed to 100 mug/L of both compounds. Methoprene exposure resulted in a concentiation-dependent increase in the metabolic androgenization ratio. Mysids exposed to nonylphenol at 10 mug/L had a significantly higher metabolic androgenization ratio. The present study indicates that energy and testosterone metabolism of mysids, as endpoints, are able to detect endocrine-disruptive activity of chemicals after short-term exposure to environmentally realistic levels of endocrine disruptors.".
- aggregation authorList BK364805.
- aggregation endPage "1296".
- aggregation issue "5".
- aggregation startPage "1289".
- aggregation volume "23".
- aggregation aggregates 1890006.
- aggregation isDescribedBy 295171.
- aggregation similarTo 03-338.
- aggregation similarTo LU-295171.