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- aggregation classification "A1".
- aggregation creator B372699.
- aggregation creator B372700.
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation date "2012".
- aggregation format "application/pdf".
- aggregation hasFormat 3039707.bibtex.
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- aggregation isPartOf urn:issn:1471-4914.
- aggregation language "eng".
- aggregation rights "I have transferred the copyright for this publication to the publisher".
- aggregation subject "Medicine and Health Sciences".
- aggregation title "ER stress-induced inflammation: does it aid or impede disease progression?".
- aggregation abstract "Different lines of research have revealed that pathways activated by the endoplasmic reticulum (ER) stress response induce sterile inflammation. When activated, all three sensors of the unfolded protein response (UPR), PERK, IRE1, and ATF6, participate in upregulating inflammatory processes. ER stress in various cells plays an important role in the pathogenesis of several diseases, including obesity, type 2 diabetes, cancer, and intestinal bowel and airway diseases. Moreover, it has been suggested that ER stress-induced inflammation contributes substantially to disease progression. However, this generalization can be challenged at least in the case of cancer. In this review, we emphasize that ER stress can either aid or impede disease progression via inflammatory pathways depending on the cell type, disease stage, and type of ER stressor.".
- aggregation authorList BK673860.
- aggregation endPage "598".
- aggregation issue "10".
- aggregation startPage "589".
- aggregation volume "18".
- aggregation aggregates 4381323.
- aggregation isDescribedBy 3039707.
- aggregation similarTo j.molmed.2012.06.010.
- aggregation similarTo LU-3039707.