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• catalog contributor b6732382.
• catalog contributor b6732383.
• catalog created "1994.".
• catalog date "1994".
• catalog date "1994.".
• catalog dateCopyrighted "1994.".
• catalog description "(cont) Cytokine-neutralizing strategies in experimental sepsis -- Implications of immunotherapy for standard care of septic patients -- LPS, recIL1, and smooth muscle cell-IL1 activate vascular cells by specific mechanisms -- Pentoxifylline modulates cytokine responses in a sepsis model -- Initial studies on the administration of C1-esterase inhibitor to patients with septic shock or with a vascular leak syndrome induced by interleukin-2 therapy -- Anti-cytokine strategies -- Gene therapy for inflammatory diseases -- Regulation of the TNF[alpha] gene -- Endotoxin and the release of tumor necrosis factor receptors -- Participation of IFN-[gamma] in the pathogenesis of LPS lethality -- Sepsis criteria and selection of patients for immunotherapy -- Protective role of interleukin-10 in endotoxin shock -- Role of tumor necrosis factor in systemic inflammatory responses in primate endotoxemia -- Cost-effectiveness of HA-1A treatment for patients with sepsis.".
• catalog description "(cont) Endotoxin-binding synthetic peptides with endotoxin-neutralizing, antibacterial and anticoagulant activities -- Hemoglobin: a newly recognized binding protein for bacterial endotoxins (LPS) -- Interference with coagulation, cell adherence, and secondary inflammatory reactions -- Studies on the inflammatory-coagulant axis in the baboon response to E. coli: regulatory roles of proteins C, S, C4bBP, and of inhibitors of tissue factor -- Contact system and sepsis -- Inhibition of extrinsic coagulation activation in endotoxemia: therapeutic implications -- Experimental and clinical evidence of leukocyte activation in trauma and sepsis -- Endotoxin-induced coagulation activation and protein C reduction in meningococcal septic shock -- Efficacy of a recombinant amino terminal fragment of bactericidal/permeability-increasing protein in rodents challenged with LPS or E. coli bacteria -- Pulmonary injury following sepsis -- Platelet-activating factor antagonists as therapeutic strategy in sepsis.".
• catalog description "Anti-endotoxin strategies -- Receptor(s) for endotoxin on mammalian cells -- Case for an endotoxic conformation -- LPS binding protein and CD14 in the LPS-dependent activation of cells -- Bactericidal/permeability-increasing protein of neutrophils is a potent antibacterial and anti-endotoxin agent in vitro and in vivo -- Septin, an activity in plasma that enables CD14-dependent recognition of LPS -- Interaction between CD14, lipopolysaccharide-binding protein and bactericidal/permeability-increasing protein during lipopolysaccharide-induced cell activation -- Biological activity of lipid A partial structures -- Anti-lipopolysaccharide core antibodies -- Lipopolysaccharide-antagonizing effects of diphosphoryl lipid A from Rhodabacter sphaeroides (Rs-DPLA) -- Prophylactic use of monophosphoryl lipid A in patients at risk for sepsis -- Rabbit CAP18-derived peptides inhibit gram-negative and gram-positive bacteria -- Phosphatidyl-inositol-linked CD14 is involved in priming of human neutrophils by lipopolysaccharide (LPS), but not in the inactivation of LPS.".
• catalog description "Includes bibliographical references and index.".
• catalog extent "xxiii, 456 p. :".
• catalog hasFormat "Bacterial endotoxins.".
• catalog identifier "0471021814".
• catalog isFormatOf "Bacterial endotoxins.".
• catalog isPartOf "Progress in clinical and biological research ; v. 388".
• catalog issued "1994".
• catalog issued "1994.".
• catalog language "eng".
• catalog publisher "New York : Wiley-Liss,".
• catalog relation "Bacterial endotoxins.".
• catalog subject "616.9/44 20".
• catalog subject "Antisepsis congresses.".
• catalog subject "Bacterial Toxins congresses.".
• catalog subject "Endotoxins Congresses.".
• catalog subject "Endotoxins congresses.".
• catalog subject "RC182.S4 I54 1993".
• catalog subject "Septic shock Congresses.".
• catalog subject "W1 PR668E v.388 1994 QW 630 I58486 1994".
• catalog tableOfContents "(cont) Cytokine-neutralizing strategies in experimental sepsis -- Implications of immunotherapy for standard care of septic patients -- LPS, recIL1, and smooth muscle cell-IL1 activate vascular cells by specific mechanisms -- Pentoxifylline modulates cytokine responses in a sepsis model -- Initial studies on the administration of C1-esterase inhibitor to patients with septic shock or with a vascular leak syndrome induced by interleukin-2 therapy -- Anti-cytokine strategies -- Gene therapy for inflammatory diseases -- Regulation of the TNF[alpha] gene -- Endotoxin and the release of tumor necrosis factor receptors -- Participation of IFN-[gamma] in the pathogenesis of LPS lethality -- Sepsis criteria and selection of patients for immunotherapy -- Protective role of interleukin-10 in endotoxin shock -- Role of tumor necrosis factor in systemic inflammatory responses in primate endotoxemia -- Cost-effectiveness of HA-1A treatment for patients with sepsis.".
• catalog tableOfContents "(cont) Endotoxin-binding synthetic peptides with endotoxin-neutralizing, antibacterial and anticoagulant activities -- Hemoglobin: a newly recognized binding protein for bacterial endotoxins (LPS) -- Interference with coagulation, cell adherence, and secondary inflammatory reactions -- Studies on the inflammatory-coagulant axis in the baboon response to E. coli: regulatory roles of proteins C, S, C4bBP, and of inhibitors of tissue factor -- Contact system and sepsis -- Inhibition of extrinsic coagulation activation in endotoxemia: therapeutic implications -- Experimental and clinical evidence of leukocyte activation in trauma and sepsis -- Endotoxin-induced coagulation activation and protein C reduction in meningococcal septic shock -- Efficacy of a recombinant amino terminal fragment of bactericidal/permeability-increasing protein in rodents challenged with LPS or E. coli bacteria -- Pulmonary injury following sepsis -- Platelet-activating factor antagonists as therapeutic strategy in sepsis.".
• catalog tableOfContents "Anti-endotoxin strategies -- Receptor(s) for endotoxin on mammalian cells -- Case for an endotoxic conformation -- LPS binding protein and CD14 in the LPS-dependent activation of cells -- Bactericidal/permeability-increasing protein of neutrophils is a potent antibacterial and anti-endotoxin agent in vitro and in vivo -- Septin, an activity in plasma that enables CD14-dependent recognition of LPS -- Interaction between CD14, lipopolysaccharide-binding protein and bactericidal/permeability-increasing protein during lipopolysaccharide-induced cell activation -- Biological activity of lipid A partial structures -- Anti-lipopolysaccharide core antibodies -- Lipopolysaccharide-antagonizing effects of diphosphoryl lipid A from Rhodabacter sphaeroides (Rs-DPLA) -- Prophylactic use of monophosphoryl lipid A in patients at risk for sepsis -- Rabbit CAP18-derived peptides inhibit gram-negative and gram-positive bacteria -- Phosphatidyl-inositol-linked CD14 is involved in priming of human neutrophils by lipopolysaccharide (LPS), but not in the inactivation of LPS.".
• catalog title "Bacterial endotoxins : basic science to anti-sepsis strategies : proceedings of the Fourth International Conference on Endotoxins (ICEA IV), held in Amsterdam, the Netherlands, August 1993 / editors, Jack Levin ... [et al.].".
• catalog type "Conference proceedings. fast".
• catalog type "text".