Matches in UGent Biblio for { <https://biblio.ugent.be/publication/1224479#aggregation> ?p ?o. }
Showing items 1 to 35 of
35
with 100 items per page.
- aggregation classification "A1".
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation creator person.
- aggregation date "2011".
- aggregation format "application/pdf".
- aggregation hasFormat 1224479.bibtex.
- aggregation hasFormat 1224479.csv.
- aggregation hasFormat 1224479.dc.
- aggregation hasFormat 1224479.didl.
- aggregation hasFormat 1224479.doc.
- aggregation hasFormat 1224479.json.
- aggregation hasFormat 1224479.mets.
- aggregation hasFormat 1224479.mods.
- aggregation hasFormat 1224479.rdf.
- aggregation hasFormat 1224479.ris.
- aggregation hasFormat 1224479.txt.
- aggregation hasFormat 1224479.xls.
- aggregation hasFormat 1224479.yaml.
- aggregation isPartOf urn:issn:0966-842X.
- aggregation language "eng".
- aggregation rights "I have transferred the copyright for this publication to the publisher".
- aggregation subject "Biology and Life Sciences".
- aggregation title "Cecal ligation and puncture: the gold standard model for polymicrobial sepsis?".
- aggregation abstract "Sepsis is a serious medical condition characterized by dysregulated systemic inflammatory responses followed by immunosuppression. To study the pathophysiology of sepsis, diverse animal models have been developed. Polymicrobial sepsis induced by cecal ligation and puncture (CLP) is the most frequently used model because it closely resembles the progression and characteristics of human sepsis. Here we summarize the role of several immune components in the pathogenesis of sepsis induced by CLP. However, several therapies proposed on the basis of promising results obtained by CLP could not be translated to the clinic. This demonstrates that experimental sepsis models do not completely mimic human sepsis. We propose several strategies to narrow the gap between experimental sepsis models and clinical sepsis, including targeting factors that contribute to the immunosuppressive phase of sepsis, and reproducing the heterogeneity of human patients.".
- aggregation authorList BK627815.
- aggregation endPage "208".
- aggregation issue "4".
- aggregation startPage "198".
- aggregation volume "19".
- aggregation aggregates 2937832.
- aggregation isDescribedBy 1224479.
- aggregation similarTo j.tim.2011.01.001.
- aggregation similarTo LU-1224479.